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Chemogenetic silencing of excitatory hippocampal neurons prevents spontaneous seizures in a mouse model for temporal lobe epilepsy
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- Conference Paper
- C3
- open access
Chemogenetic silencing of excitatory hippocampal neurons prevents spontaneous seizures in a mouse model for temporal lobe epilepsy
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- Conference Paper
- C3
- open access
Chemogenetic silencing of excitatory hippocampal neurons prevents spontaneous seizures in a mouse model for temporal lobe epilepsy
-
- Conference Paper
- C3
- open access
Chemogenetic silencing of excitatory hippocampal neurons prevents spontaneous seizures in a mouse model for temporal lobe epilepsy
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Chemogenetic silencing of excitatory hippocampal neurons prevents spontaneous seizures in a mouse model for temporal lobe epilepsy
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- Conference Paper
- C3
- open access
The effect of chemogenetic inhibition of excitatory neurons on hippocampal excitability and seizure frequency in a rat model
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- Conference Paper
- C3
- open access
The effect of chemogenetic inhibition of excitatory neurons on hippocampal excitability and seizure frequency in a rat model
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Uric acid is released in the brain during seizure activity and increases severity of seizures in a mouse model for acute limbic seizures
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Vagus nerve stimulation applied with a rapid cycle has more profound influence on hippocampal electrophysiology than a standard cycle
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Modulation of hippocampal activity by vagus nerve stimulation in freely moving rats
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The effect of neuropeptide FF in the amygdala kindling model
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The systemic kainic acid rat model of temporal lobe epilepsy: long-term EEG monitoring
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In search of optimal DBS paradigms to treat epilepsy: bilateral versus unilateral hippocampal stimulation in a rat model for temporal lobe epilepsy
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Neuropeptide FF and prolactin-releasing peptide decrease cortical excitability through activation of NPFF receptors
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Vagus nerve stimulation has antidepressant effects in the kainic acid model for temporal lobe epilepsy