Show
Sort by
-
Non-classical ProIL-1beta activation during mammary gland infection is pathogen-dependent but caspase-1 independent
-
- Journal Article
- A1
- open access
Non-classical ProIL-1beta activation during mammary gland infection is pathogen-dependent but caspase-1 independent
-
Simultaneous targeting of IL-1 and IL-18 is required for protection against inflammatory and septic shock
-
- Journal Article
- A1
- open access
Escherichia coli induces bovine neutrophil cell death independent from caspase-3/-7/-1, but with phosphatidylserine exposure prior to membrane rupture
-
An inactivating caspase-11 passenger mutation muddles sepsis research
-
The in vivo role of executioner caspase-3 and -7 in the Escherichia coli-infected mammary gland
-
In vitro characterization of bovine neutrophil cell death following Escherichia coli phagocytosis
-
Role of caspase-7 during early involution of the Escherichia coli- or the Staphylococcus aureus-infected mammary gland
-
Caspase substrates: easily caught in deep waters?
-
Proteome-wide substrate analysis indicates substrate exclusion as a mechanism to generate caspase-7 versus caspase-3 specificity