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- 2015
- Essential versus accessory aspects of cell death: recommendations of the NCCD 2015
- Endoplasmic reticulum stress induces ligand-independent TNFR1-mediated necroptosis in L929 cells
- 2014
- MLKL compromises plasma membrane integrity by binding to phosphatidylinositol phosphates
- Deficiency in the mitochondrial apoptotic pathway reveals the toxic potential of autophagy under ER stress conditions
- RIPK1 promotes death receptor-independent caspase-8-mediated apoptosis under unresolved ER stress conditions
- Depletion of RIPK3 or MLKL blocks TNF-driven necroptosis and switches towards a delayed RIPK1 kinase-dependent apoptosis
- 2012
- RIP1 is required for IAP inhibitor-mediated sensitization of childhood acute leukemia cells to chemotherapy-induced apoptosis
- Intermediate domain of receptor-interacting protein kinase 1 (RIPK1) determines switch between necroptosis and RIPK1 kinase-dependent apoptosis
- The role of the IAP E3 ubiquitin ligases in regulating pattern-recognition receptor signalling
- The unfolded protein response at the crossroads of cellular life and death during endoplasmic reticulum stress