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Crosstalk interactions between transcription factors ERRα and PPARα assist PPARα-mediated gene expression
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RIPK1 is dispensable for cell death regulation in β-cells during hyperglycemia
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Inhibition of RIPK1 kinase does not affect diabetes development : beta-cells survive RIPK1 activation
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The hepatocyte IKK:NF-kB axis promotes liver steatosis by stimulating de novo lipogenesis and cholesterol synthesis