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Clostridium perfringens strains proliferate to high counts in the broiler small intestinal tract, in accordance with necrotic lesion severity, and sporulate in the distal intestine
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NanI sialidase contributes to toxin expression and host cell binding of Clostridium perfringens type G strain CP56 in vitro
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Protein truncating variants of colA in clostridium perfringens type G strains
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Research note: The administration schedule of coccidia is a major determinant in broiler necrotic enteritis models
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C. perfringens challenge reduces matrix metalloproteinase activity in the jejunal mucosa of Eimeria-infected broiler chickens
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Incidence and associated risk factors of necrotic enteritis in Belgian layer pullet flocks
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Spotlight on avian pathology : untangling contradictory disease descriptions of necrotic enteritis and necro-haemorrhagic enteritis in broilers
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A comparative study on the use of selective media for the enumeration of Clostridium perfringens in poultry faeces
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Rapid growth predisposes broilers to necrotic enteritis
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Elevated faecal ovotransferrin concentrations are indicative for intestinal barrier failure in broiler chickens