Ghent University Academic Bibliography

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Hospital mortality in COVID-19 patients in Belgium treated with statins, ACE inhibitors and/or ARBs

Geert Byttebier, Luc Belmans (UGent) , Myriam Alexander, Bo E. H. Saxberg, Bart De Spiegeleer (UGent) , Anton De Spiegeleer (UGent) , Nick Devreker, Jens Van Praet (UGent) , Karolien Vanhove, Reinhilde Reybrouck, et al.

(2021) HUMAN VACCINES & IMMUNOTHERAPEUTICS. 17(9). p.2841-2850
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- Journal Article
- A1
- open access
Macrophages in obesity and non-alcoholic fatty liver disease : crosstalk with metabolism

Sander Lefere (UGent) and Frank Tacke

(2019) JHEP REPORTS. 1(1). p.30-43
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- PhD Thesis
Angiogenesis and macrophages as diagnostic and therapeutic targets in non-alcoholic fatty liver disease

Sander Lefere (UGent)

(2019)
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The lung environment controls alveolar macrophage metabolism and responsiveness in type 2 inflammation

Freya Svedberg (UGent) , Sheila L Brown, Maria Z Krauss, Laura Campbell, Catherine Sharpe, Maryam Clausen, Gareth J Howell, Howard Clark, Jens Madsen, Christopher M Evans, et al.

(2019) NATURE IMMUNOLOGY. 20(5). p.571-580
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A bidirectional crosstalk between iNKT cells and adipocytes mediated by leptin modulates susceptibility for T cell mediated hepatitis

Koen Venken (UGent) , Sylvie Seeuws (UGent) , Lennart Zabeau (UGent) , Peggy Jacques (UGent) , Tine Decruy (UGent) , Julie Coudenys (UGent) , Eveline Verheugen (UGent) , Fien Windels (UGent) , Dominiek Catteeuw (UGent) , Michael Drennan (UGent) , et al.

(2014) JOURNAL OF HEPATOLOGY. 60(1). p.175-182

Academic Bibliography

Hospital mortality in COVID-19 patients in Belgium treated with statins, ACE inhibitors and/or ARBs

Macrophages in obesity and non-alcoholic fatty liver disease : crosstalk with metabolism

Angiogenesis and macrophages as diagnostic and therapeutic targets in non-alcoholic fatty liver disease

The lung environment controls alveolar macrophage metabolism and responsiveness in type 2 inflammation

A bidirectional crosstalk between iNKT cells and adipocytes mediated by leptin modulates susceptibility for T cell mediated hepatitis

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