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Study of NFAT5 physiology in Duchenne muscular dystrophy fibroblasts : provisionary explanation for the permanent fibrosis formation in Duchenne muscular dystrophy
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Inflammatory cell-derived CXCL3 promotes pancreatic cancer metastasis through a novel myofibroblast-hijacked cancer escape mechanism
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Mathematical modelling of the mechano-electric coupling in the human cardiomyocyte electrically connected with fibroblasts
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Duchenne muscular dystrophy fibrosis potentially explained by abnormal NFAT5 physiology in Duchenne muscular dystrophy fibroblasts
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- Journal Article
- A1
- open access
Involvement of the extracellular matrix proteins periostin and tenascin C in nasal polyp remodeling by regulating the expression of MMPs
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- Journal Article
- A1
- open access
Multiparametric analysis of geometric features of fibrotic textures leading to cardiac arrhythmias
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Metabolic modulation during intestinal fibrosis
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- Journal Article
- A1
- open access
Abnormal NFAT5 physiology in Duchenne muscular dystrophy fibroblasts as a putative explanation for the permanent fibrosis formation in Duchenne muscular dystrophy
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The role of the peritoneal microenvironment in the pathogenesis of colorectal peritoneal carcinomatosis
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Targeting the tumor microenvironment in colorectal peritoneal metastases