Ghent University Academic Bibliography

Journal Article
A1
open access

Transient nuclear lamin A/C accretion aids in recovery from vapor nanobubble-induced permeabilisation of the plasma membrane

Gaëlle Houthaeve, Gerardo García-Díaz Barriga, Stephan Stremersch (UGent) , Herlinde De Keersmaecker (UGent) , Juan Fraire (UGent) , Jo Vandesompele (UGent) , Pieter Mestdagh (UGent) , Stefaan De Smedt (UGent) , Kevin Braeckmans (UGent) and Winnok De Vos (UGent)

(2022) CELLULAR AND MOLECULAR LIFE SCIENCES. 79(1).

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Loss of nuclear envelope integrity in aging and disease

Joke Robijns, Gaëlle Houthaeve, Kevin Braeckmans (UGent) and Winnok De Vos (UGent)

(2018) International Review of Cell and Molecular Biology, vol. 336. 336. p.205-222

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Journal Article
A1
open access

Sustained accumulation of prelamin A and depletion of lamin A/C both cause oxidative stress and mitochondrial dysfunction but induce different cell fates

Tom Sieprath (UGent) , Tobias Corne (UGent) , Marco Nooteboom, Charlotte Grootaert (UGent) , Andreja Rajkovic (UGent) , Benjamin Buysschaert (UGent) , Joke Robijns, Jos LV Broers, Frans CS Ramaekers, Werner JH Koopman, et al.

(2015) NUCLEUS. 6(3). p.236-246

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Lamins as mediators of oxidative stress

Tom Sieprath (UGent) , Rabih Darwiche (UGent) and Winnok De Vos (UGent)

(2012) BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS. 421(4). p.635-639

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Repetitive disruptions of the nuclear envelope invoke temporary loss of cellular compartmentalization in laminopathies

Winnok De Vos (UGent) , Frederik Houben, Miriam Kamps, Ashraf Malhas, Fons Verheyen, Juliën Cox, Erik Manders (UGent) , Valerie LRM Verstraeten, Maurice AM van Steensel, Carlo LM Marcelis, et al.

(2011) HUMAN MOLECULAR GENETICS. 20(21). p.4175-4186

Academic Bibliography

Transient nuclear lamin A/C accretion aids in recovery from vapor nanobubble-induced permeabilisation of the plasma membrane

Loss of nuclear envelope integrity in aging and disease

Sustained accumulation of prelamin A and depletion of lamin A/C both cause oxidative stress and mitochondrial dysfunction but induce different cell fates

Lamins as mediators of oxidative stress

Repetitive disruptions of the nuclear envelope invoke temporary loss of cellular compartmentalization in laminopathies

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