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Functional knockout of the soluble guanylate cyclase alpha 1 subunit leads to gender-specific hypertension while retaining sensitivity to nitric oxide
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Blood pressure in mice with a targeted deletion of the soluble guanylyl cyclase alpha 1 subunit
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Functional knockout of the soluble guanylate cyclase alpha 1 subunit leads to gender-specific hypertension while retaining sensitivity to nitric oxide
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Gender-specific hypertension in mice deficient in the alpha(1) subunit of soluble guanylate cyclase