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- Journal Article
- A1
- open access
RIPK1 kinase-dependent inflammation and cell death contribute to the pathogenesis of COPD
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Apoptotic cell death in disease : current understanding of the NCCD 2023
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- Journal Article
- A1
- open access
Molecular mechanisms of cell death : recommendations of the Nomenclature Committee on Cell Death 2018
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A real-time fluorometric method for the simultaneous detection of cell death type and rate
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- Journal Article
- A2
- open access
Molecular crosstalk between apoptosis, necroptosis, and survival signaling
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- Journal Article
- A1
- open access
Essential versus accessory aspects of cell death: recommendations of the NCCD 2015
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- Journal Article
- A1
- open access
Depletion of RIPK3 or MLKL blocks TNF-driven necroptosis and switches towards a delayed RIPK1 kinase-dependent apoptosis
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Intermediate domain of receptor-interacting protein kinase 1 (RIPK1) determines switch between necroptosis and RIPK1 kinase-dependent apoptosis
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TNF-induced necroptosis in L929 cells is tightly regulated by multiple TNFR1 complex I and II members
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cIAP1 and TAK1 protect cells from TNF-induced necrosis by preventing RIP1/RIP3-dependent reactive oxygen species production