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- Journal Article
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- open access
Overexpression of alpha(1)-acid glycoprotein in transgenic mice leads to sensitisation to acute colitis.
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A mediator role for metallothionein in tumor necrosis factor-induced lethal shock
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Detection, characterisation and purification of a murine liver factor capable of desensitising towards the lethal activity of tumour necrosis factor.
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Differential response of alpha(2)-macroglobulin-deficient mice in models of lethal TNF-induced inflammation.
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- Journal Article
- A1
- open access
The major acute-phase protein, serum amyloid P component, in mice is not involved in endogenous resistance against tumor necrosis factor alpha-induced lethal hepatitis, shock, and skin necrosis.
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Tumor necrosis factor-induced lethal hepatitis: pharmacological intervention with verapamil, tannic acid, picotamide and K76COOH.
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Identification of a locus on distal mouse chromosome 12 that controls resistance to tumor necrosis factor-induced lethal shock.
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The role of complement activation in tumour necrosis factor-induced lethal hepatitis.
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Caspase-1 is not involved in experimental hepatitis in mouse.
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Activation of caspases in lethal experimental hepatitis and prevention by acute phase proteins.