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- Journal Article
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The impact of hepatocyte-specific deletion of hypoxia-inducible factors on the development of polymicrobial sepsis with focus on GR and PPARĪ± function
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Reprogramming of glucocorticoid receptor function by hypoxia
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- Miscellaneous
- open access
Turning a pathogen protein into a therapeutic tool for sepsis
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- Journal Article
- A1
- open access
ZBTB32 performs crosstalk with the glucocorticoid receptor and is crucial in glucocorticoid responses to starvation
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Combined glucocorticoid resistance and hyperlactatemia contributes to lethal shock in sepsis
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- Journal Article
- A1
- open access
Hepatic PPARĪ± function and lipid metabolic pathways are dysregulated in polymicrobial sepsis
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Glucocorticoids limit lipopolysaccharide-induced lethal inflammation by a double control system
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- Journal Article
- A1
- open access
Zinc inhibits lethal inflammatory shock by preventing microbe-induced interferon signature in intestinal epithelium