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Antinuclear antibodies following infliximab treatment in patients with rheumatoid arthritis or spondylarthropathy
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Changes in rheumatoid factor reflect the inflammatory response (CRP and ESR) to infliximab treatment
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Increase in lymphoid follicles and leukocyte adhesion molecules emphasizes a role for the gut in spondyloarthropathy pathogenesis.
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Macrophages expressing the scavenger receptor CD163: a link between immune alterations of the gut and synovial inflammation in spondyloarthropathy.
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Influence of the gut and cytokine patterns in spondyloarthropathy.
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Immunomodulatory effects of anti-tumor necrosis factor α therapy on synovium in spondylarthropathy: histologic findings in eight patients from an open-label pilot study
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The proportion of Th1 cells, which prevail in gut mucosa, is decreased in inflammatory bowel syndrome.
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Gut mucosal T cell lines from ankylosing spondylitis patients are enriched with alpha E beta 7 integrin.
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Impaired Th1 cytokine production in spondyloarthropathy is restored by anti-TNFα
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Flow cytometric analysis of gut mucosal lymphocytes supports an impaired Th1 cytokine profile in spondyloarthropathy.
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Comparative study of the synovial histology in rheumatoid arthritis, spondyloarthropathy, and osteoarthritis: influence of disease duration and activity
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Chemical agents and enzymes used for the extraction of gut lymphocytes influence flow cytometric detection of T cell surface markers.
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Focal up-regulation of E-cadherin-catenin complex in inflamed bowel mucosa but reduced expression in ulcer associated cell lineage.
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Subclinical gut inflammation in spondyloarthropathy patients is associated with upregulation of the E-cadherin/catenin complex.
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Opportunities for immune modulation in the spondyloarthropathies with special reference to gut inflammation.
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Crohn's disease and ulcerative colitis show different distribution patterns of CD83 positive dendritic cells
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T cell cytokines in gut mucosa of patients with spondyloarthropathy reflect immune alterations in Crohn's disease
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Inflammatory bowel disease and subclinical gut inflammation in spondyloarthropathy patients are associated with increased expression of the E-cadherin/catenin complex
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Intestinal mucosa of patients with spondyloarthropathy is enriched with T cells carrying alpha E beta 7 integrin, even in the absence of histologically defined inflammation
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Effects of chemical agents and enzymes on lymphocyte surface markers
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Inflammatory bowel disease and subclinical gut inflammation in spondyloarthropathy patients are associated with increased expression of the E-cadherin/catenin complex
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Upregulation of E-cadherin and associated molecules (a-, ß- and y-catenins) in inflamed bowel mucosa
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Increased expression of E 7 integrin in uninflamed mucosa of patients with spondyloarthropathy
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Effects of chemical agents and enzymes on the detection of lymphocyte surface markers.
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Isolation of colonie mucosal lamina propria and intra-epithelial lymphocytes : mechanical versus enzymatic isolation
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Altered expression of ocEß7 integrin on intra-epithelial and lamina propria lymphocytes in patients with Crohn's disease
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A comparative phenotypical analysis of rheumatoid nodules and rheumatoid synovium with special reference to adhesion molecules and activation markers.