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- Conference Paper
- C3
- open access
Reduced metallothionein expression in colonic Crohn's disease: evidence for a new disease-modifying gene
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Reduced metallothionein expression in colonic Crohn's disease: evidence for it new disease-modifying gene
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Altered gut transcriptome in spondyloarthropathy
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Alternative method to identify susceptibility genes for Crohn's disease through transcriptome analysis
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CARD15 polymorphisms are associated with anti-Saccharomyces cerevisiae antibodies in caucasian Crohn's disease patients
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Colon mucosa of patients both with spondyloarthritis and Crohn's disease is enriched with macrophages expressing the scavenger receptor CD163
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- Journal Article
- A1
- open access
CARD15 gene polymorphisms in patients with spondyloarthropathies identify a specific phenotype previously related to Crohn's disease
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- Journal Article
- A1
- open access
Radiological sacroiliitis, a hallmark of spondylitis, is linked with CARD15 gene polymorphisms in patients with Crohn's disease
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Crohn's disease patients with CARD15 variants have higher risk of developing axial joint involvement
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CARD15 mutations in patients with spondyloarthropathy are linked with disease progression and evolution to Crohn's disease
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Association between ASCA-positivity and carrier of a CARD-15 mutation in patients with Crohn's disease
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Gut inflammation and spondyloarthropathies.
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Increase in lymphoid follicles and leukocyte adhesion molecules emphasizes a role for the gut in spondyloarthropathy pathogenesis.
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Macrophages expressing the scavenger receptor CD163: a link between immune alterations of the gut and synovial inflammation in spondyloarthropathy.
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Immunomodulatory effects of anti-tumor necrosis factor α therapy on synovium in spondylarthropathy: histologic findings in eight patients from an open-label pilot study
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The proportion of Th1 cells, which prevail in gut mucosa, is decreased in inflammatory bowel syndrome.
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Gut mucosal T cell lines from ankylosing spondylitis patients are enriched with alpha E beta 7 integrin.
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Comparative study of the synovial histology in rheumatoid arthritis, spondyloarthropathy, and osteoarthritis: influence of disease duration and activity
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Chemical agents and enzymes used for the extraction of gut lymphocytes influence flow cytometric detection of T cell surface markers.
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Focal up-regulation of E-cadherin-catenin complex in inflamed bowel mucosa but reduced expression in ulcer associated cell lineage.
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Subclinical gut inflammation in spondyloarthropathy patients is associated with upregulation of the E-cadherin/catenin complex.
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Spondyloarthropathy is underestimated in inflammatory bowel disease: Prevalence and HLA association.
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High frequency of 'missed' HLA-B loci by serologic determination in patients with spondyloarthropathy and chronic gut inflammation
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Crohn's disease and ulcerative colitis show different distribution patterns of CD83 positive dendritic cells
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T cell cytokines in gut mucosa of patients with spondyloarthropathy reflect immune alterations in Crohn's disease
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Inflammatory bowel disease and subclinical gut inflammation in spondyloarthropathy patients are associated with increased expression of the E-cadherin/catenin complex
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Intestinal mucosa of patients with spondyloarthropathy is enriched with T cells carrying alpha E beta 7 integrin, even in the absence of histologically defined inflammation
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Effects of chemical agents and enzymes on lymphocyte surface markers
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Enrichment of T cells carrying 137 integrins in inflamed synovial tissue from patients with early spondyloarthropathy, compared to rheumatoid arthritis
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Altered expression of ocEß7 integrin on intra-epithelial and lamina propria lymphocytes in patients with Crohn's disease
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Distinctive activated cellular subsets in colon from patients with Crohn's disease and ulcerative colitis
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Bone and joint diseases in inflammatory bowel disease
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Bowel inflammation and the spondyloarthropathies.
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A comparative phenotypical analysis of rheumatoid nodules and rheumatoid synovium with special reference to adhesion molecules and activation markers.
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Immunopathology of the enteropathies
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The immunological basis of inflammatory bowel disease
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The gut associated addressins: Lymphocyte homing in the gut.
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The gut associated adressins : lymphocyte homing in the gut
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THE ROLE OF T-CELLS IN RHEUMATOID-ARTHRITIS.
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T cell receptor Vbeta usage in rheumatoid nodules - marked oligoclonality among DL-2 expanded lymphocytes.