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Mechanisms underlying the functional cooperation between PPARα and GRα to attenuate inflammatory responses
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The autophagy receptor SQSTM1/p62 mediates anti-inflammatory actions of the selective NR3C1/glucocorticoid receptor modulator compound A (CpdA) in macrophages
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Compound A influences gene regulation of the dexamethasone-activated glucocorticoid receptor by alternative cofactor recruitment
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Chromatin recruitment of activated AMPK drives fasting response genes co-controlled by GR and PPARα