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An alphaherpesvirus exploits antimicrobial beta-defensins to initiate respiratory tract infection
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Equine herpesvirus 1 infection orchestrates the expression of chemokines in equine respiratory epithelial cells
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Unravelling the first key steps in equine herpesvirus type 5 (EHV5) pathogenesis using ex vivo and in vitro equine models
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Pollens destroy respiratory epithelial cell anchors and drive alphaherpesvirus infection
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Beyond gut instinct : metabolic short-chain fatty acids moderate the pathogenesis of alphaherpesviruses
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Deoxynivalenol, but not fumonisin B1, aflatoxin B1 or diesel exhaust particles disrupt integrity of the horse's respiratory epithelium and predispose it for equine herpesvirus type 1 infection
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Equine herpesvirus 1 bridles T lymphocytes to reach its target organs
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Abortigenic but not neurotropic equine herpes virus 1 modulates the interferon antiviral defense
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Access to a main alphaherpesvirus receptor, located basolaterally in the respiratory epithelium, is masked by intercellular junctions
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CCL2 and CCL5 driven attraction of CD172a+ monocytic cells during an equine herpesvirus type 1 (EHV-1) infection in equine nasal mucosa and the impact of two migration inhibitors, rosiglitazone (RSG) and quinacrine (QC)