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Enterotoxigenic Escherichia coli induce pro-inflammatory responses in porcine intestinal epithelial cells
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- Conference Paper
- C3
- open access
The food contaminant fumonisin B1 reduces the maturation of porcine CD11R1+ intestinal dendritic cells, resulting in a reduced efficiency of oral immunisation and a prolonged intestinal ETEC infection
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Rectal inoculation of sheep with E. coli O157:H7 results in persistent infection in the absence of a protective immune response
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Enterotoxigenic Escherichia coli (K88) induce proinflammatory responses in porcine intestinal epithelial cells
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The food contaminant fumonisin B1 reduces the maturation of porcine CD11R1+ intestinal antigen presenting cells and antigen-specific immune responses, leading to a prolonged intestinal ETEC infection
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Targeting of Escherichia coli F4 fimbriae to Fcγ receptors enhances the maturation of porcine dendritic cells
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The food contaminant fumonisin B1 reduces the maturation of porcine CD11R1+ intestinal dendritic cells, resulting in a reduced efficiency of oral immunisation and a prolonged intestinal ETEC infection
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Selective antigen targeting to FcgR enhances the funtional maturation of porcine dendritic cells
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Maltose-binding protein (MBP) is a potential carrier for oral immunizations
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T cell activation after priming by immune complex matured porcine monocytic-derived dendritic cells