Show
Sort by
-
MiR-34a deficiency accelerates medulloblastoma formation in vivo
-
- Journal Article
- A1
- open access
A Cre-conditional MYCN-driven neuroblastoma mouse model as an improved tool for preclinical studies
-
MYCN and ALKF1174L are sufficient to drive neuroblastoma development from neural crest progenitor cells
-
- Journal Article
- A1
- open access
BET bromodomain protein inhibition is a therapeutic option for medulloblastoma
-
LIN28B induces neuroblastoma and enhances MYCN levels via let-7 suppression
-
Targeted expression of mutated ALK induces neuroblastoma in transgenic mice
-
Pharmacological activation of the p53 pathway by nutlin-3 exerts anti-tumoral effects in medulloblastomas
-
Lysine-specific demethylase 1 restricts hematopoietic progenitor proliferation and is essential for terminal differentiation
-
ALK(F1174L) is a driving oncogene of neuroblastoma in transgenic mice
-
Pharmacological activation of the p53 pathway by nutlin-3 inhibits medulloblastoma cell proliferation in vitro and in vivo