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ALK signaling primes the DNA damage response sensitizing ALK-driven neuroblastoma to therapeutic ATR inhibition
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ALK fusion NSCLC oncogenes promote survival and inhibit NK cell responses via SERPINB4 expression
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Loss of RET promotes mesenchymal identity in neuroblastoma cells
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ATR inhibition enables complete tumour regression in ALK-driven NB mouse models
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11q deletion or ALK activity curbs DLG2 expression to maintain an undifferentiated state in neuroblastoma