- Different pathways mediate cytochrome c release after photodynamic therapy with hypericin.
- The somatostatin analogue TT-232 induces apoptosis in A431 cells - Sustained activation of stress-activated kinases and inhibition of signalling to extracellular signal-regulated kinases.
- In vitro and in vivo evaluation of hypericin for photodynamic therapy of equine sarcoids.
A novel endogenous PP2C-like phosphatase dephosphorylates casein kinase II-phosphorylated Physarum fragmin.
2000) BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS. 279(2). p.438-444 Mark(
- p38 mitogen-activated protein kinase regulates a novel, caspase-independent pathway for the mitochondrial cytochrome c release in ultraviolet B radiation-induced apoptosis.
- The activation of the c-Jun N-terminal kinase and p38 mitogen-activated protein kinase signaling pathways protects HeLa cells from apoptosis following photodynamic therapy with hypericin.
- Hypericin-induced photosensitization of HeLa cells leads to apoptosis or necrosis - Involvement of cytochrome c and procaspase-3 activation in the mechanism of apoptosis.
- Microfilament Dynamics - Regulation of Actin Polymerization by Actin-Fragmin Kinase and Phosphatases.
- Molecular clotting, expression an characterization of PTPA, a protein that activates the tyrosyl phosphate activity of protein phosphatase 2A.
- TUMOR-NECROSIS-FACTOR STIMULATES MULTIPLE SERINE THREONINE PROTEIN-KINASES IN SWISS 3T3 AND L929 CELLS - IMPLICATION OF CASEIN KINASE-2 AND EXTRACELLULAR SIGNAL-REGULATED KINASES IN THE TUMOR-NECROSIS-FACTOR SIGNAL TRANSDUCTION PATHWAY.