Ghent University Academic Bibliography

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The Alzheimer susceptibility gene BIN1 induces isoform-dependent neurotoxicity through early endosome defects

Erwan Lambert, Orthis Saha, Bruna Soares Landeira, Ana Raquel Melo de Farias, Xavier Hermant, Arnaud Carrier, Alexandre Pelletier, Johanna Gadaut, Lindsay Davoine, Cloe Dupont, et al.

(2022) ACTA NEUROPATHOLOGICA COMMUNICATIONS. 10(1).
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Alternative glycosylation controls endoplasmic reticulum dynamics and tubular extension in mammalian cells

Despoina Kerselidou, Bushra Saeed Dohai, David R. Nelson, Sarah Daakour, Nicolas De Cock, Zahra Al Oula Hassoun, Dae-Kyum Kim, Julien Olivet, Diana C. El Assal, Ashish Jaiswal, et al.

(2021) SCIENCE ADVANCES. 7(19).
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Male-specific epistasis between WWC1 and TLN2 genes is associated with Alzheimer's disease

Elena S Gusareva, Jean-Claude Twizere, Kristel Sleegers, Pierre Dourlen, Jose F Abisambra, Shelby Meier, Ryan Cloyd, Blaine Weiss, Bart Dermaut (UGent) , Kyrylo Bessonov, et al.

(2018) NEUROBIOLOGY OF AGING. 72.
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Developmental expression of 4-repeat-Tau induces neuronal aneuploidy in Drosophila tauopathy models

Nicolas Malmanche, Pierre Dourlen, Marc Gistelinck, Florie Demiautte, Nichole Link, Cloé Dupont, Lies Vanden Broeck, Elisabeth Werkmeister, Philippe Amouyel, Antonino Bongiovanni, et al.

(2017) SCIENTIFIC REPORTS. 7.
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ADAM30 downregulates APP-linked defects through cathepsin D activation in Alzheimer's disease

Florent Letronne, Geoffroy Laumet, Anne-Marie Ayral, Julien Chapuis, Florie Demiautte, Mathias Laga (UGent) , Michel E Vandenberghe, Nicolas Malmanche, Florence Leroux, Fanny Eysert, et al.

(2016) EBIOMEDICINE. 9. p.278-292
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Functional complementation in Drosophila to predict the pathogenicity of TARDBP variants: evidence for a loss-of-function mechanism.

Lies Vanden Broeck, Gernot Kleinberger, Julien Chapuis, Marc Gistelinck, Philippe Amouyel, Christine Van Broeckhoven, Jean-Charles Lambert, Patrick Callaerts and Bart Dermaut (UGent)

(2015) NEUROBIOLOGY OF AGING. 36(2). p.1121-1129
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Tau phosphorylation regulates the interaction between BIN1's SH3 domain and Tau's proline-rich domain

Yoann Sottejeau, Alexis Bretteville, François-Xavier Cantrelle, Nicolas Malmanche, Florie Demiaute, Tiago Mendes, Charlotte Delay, Harmony Alves Dos Alves, Amandine Flaig, Peter Davies, et al.

(2015) ACTA NEUROPATHOLOGICA COMMUNICATIONS. 3.
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Genome-wide association interaction analysis for Alzheimer's disease

Elena S Gusareva, Minerva M Carrasquillo, Celine Bellenguez, Elise Cuyvers, Samuel Colon, Neill R Graff-Radford, Ronald C Petersen, Dennis W Dickson, Jestinah M Mahachie John, Kyrylo Bessonov, et al.

(2014) NEUROBIOLOGY OF AGING. 35(11). p.2436-2443
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TDP-43 loss-of-function causes neuronal loss due to defective steroid receptor-mediated gene program switching in Drosophila

Lies Vanden Broeck, Marina Naval-Sánchez, Yoshitsugu Adachi, Danielle Diaper, Pierre Dourlen, Julien Chapuis, Gernot Kleinberger, Marc Gistelinck, Christine Van Broeckhoven, Jean-Charles Lambert, et al.

(2013) CELL REPORTS. 3(1). p.160-172
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Drosophila models of tauopathies : what have we learned?

Marc Gistelinck, Jean-Charles Lambert, Patrick Callaerts, Bart Dermaut (UGent) and Pierre Dourlen

(2012) INTERNATIONAL JOURNAL OF ALZHEIMER'S DISEASE. 2012.

Academic Bibliography

The Alzheimer susceptibility gene BIN1 induces isoform-dependent neurotoxicity through early endosome defects

Alternative glycosylation controls endoplasmic reticulum dynamics and tubular extension in mammalian cells

Male-specific epistasis between WWC1 and TLN2 genes is associated with Alzheimer's disease

Developmental expression of 4-repeat-Tau induces neuronal aneuploidy in Drosophila tauopathy models

ADAM30 downregulates APP-linked defects through cathepsin D activation in Alzheimer's disease

Functional complementation in Drosophila to predict the pathogenicity of TARDBP variants: evidence for a loss-of-function mechanism.

Tau phosphorylation regulates the interaction between BIN1's SH3 domain and Tau's proline-rich domain

Genome-wide association interaction analysis for Alzheimer's disease

TDP-43 loss-of-function causes neuronal loss due to defective steroid receptor-mediated gene program switching in Drosophila

Drosophila models of tauopathies : what have we learned?

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