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Prevalence, clinical characteristics and prognosis of GATA2-related myelodysplastic syndromes (MDS) in children and adolescents
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Clonal mutational landscape of childhood myelodysplastic syndromes
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Somatic genetic and epigenetic architecture of myelodysplastic syndromes arising from GATA2 deficiency
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Spliceosomal gene aberrations are rare, coexist with oncogenic mutations, and are unlikely to exert a driver effect in childhood MDS and JMML
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Mutations of the spliceosome complex genes occur in adult patients but are very rare in children with myeloid neoplasia
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Refractory Cytopenia in Childhood (RCC) with normal karyotype is unlikely to progress to advanced MDS under a watch and wait strategy