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Open ADAMTS13, induced by antibodies, is a biomarker for subclinical immune-mediated thrombotic thrombocytopenic purpura
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- Journal Article
- A1
- open access
Major changes of von Willebrand factor multimer distribution in cirrhotic patients with stable disease or acute decompensation
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Blood platelet biochemistry
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Inhibition of von Willebrand factor-platelet glycoprotein Ib interaction prevents and reverses symptoms of acute acquired thrombotic thrombocytopenic purpura in baboons
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Platelets at work in primary hemostasis
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Local elongation of endothelial cell-anchored von Willebrand factor strings precedes ADAMTS13 protein-mediated proteolysis
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Thrombotic thrombocytopenic purpura directly linked with ADAMTS13 inhibition in the baboon (Papio ursinus)
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ADAMTS13 in health and disease
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Mutation of the H-bond acceptor S119 in the ADAMTS13 metalloprotease domain reduces secretion and substrate turnover in a patient with congenital thrombotic thrombocytopenic purpura
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Inherited traits affecting platelet function
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Inhibition of platelet glycoprotein Ib and its antithrombotic potential
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ADAMTS13 activity to antigen ratio in physiological and pathological conditions associated with an increased risk of thrombosis
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Activation of αIIbβ3 is a sufficient but also an imperative prerequisite for activation of α2β1 on platelets
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Decreased ADAMTS-13 (A disintegrin-like and metalloprotease with thrombospondin type 1 repeats) is associated with a poor prognosis in sepsis-induced organ failure
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Different functional active conformations of integrin a2b1 are induced dependent on the way platelets are activated.
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Two functional active conformations of the integrin α2β1, depending on activation condition and cell type
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Platelet antigens and their function
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New approaches for antithrombotic antiplatelet therapies
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Selection of specific peptides by phage display technology as affinity ligands to purify human lactoferrin from milk with expanded bed adsorption