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Erectile dysfunction in heme-deficient nitric oxide-unresponsive soluble guanylate cyclase knock-in mice
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- Journal Article
- A1
- open access
Decreased soluble guanylate cyclase contributes to cardiac dysfunction induced by chronic doxorubicin treatment in mice
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- Journal Article
- A1
- open access
The ability of nitric oxide to lower intraocular pressure is dependent on guanylyl cyclase
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Distinct myocardial mechanisms underlie cardiac dysfunction in endotoxemic male and female mice
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- Journal Article
- A1
- open access
Nitric oxide regulates skeletal muscle fatigue, fiber type, microtubule organization, and mitochondrial ATP synthesis efficiency through cGMP-dependent mechanisms
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Androgen-sensitive hypertension associated with soluble guanylate cyclase-α₁ deficiency is mediated by 20-HETE
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- Journal Article
- A1
- open access
Cardiovascular and pharmacological implications of haem-deficient NO-unresponsive soluble guanylate cyclase knock-in mice
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Genetic modification of hypertension by sGCα₁
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Brown adipose tissue blood flow and mass in obesity: a contrast ultrasound study in mice
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SERCA Cys674 sulphonylation and inhibition of L-type Ca2+ influx contribute to cardiac dysfunction in endotoxemic mice, independent of cGMP synthesis