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- 2015
- Sustained accumulation of prelamin A and depletion of lamin A/C both cause oxidative stress and mitochondrial dysfunction but induce different cell fates
- 2011
- Repetitive disruptions of the nuclear envelope invoke temporary loss of cellular compartmentalization in laminopathies
- Multiplexed profiling of secreted proteins for the detection of potential space biomarkers
- 2010
- Increased plasticity of the nuclear envelope and hypermobility of telomeres due to the loss of A-type lamins