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Protective role of soluble guanylate cyclase on the outcome of cardiac arrest and resuscitation in mice
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Soluble guanylate cyclase is essential for the pulmonary but not the systemic vasodilator response to NO and limits pulmonary vascular remodeling in response to chronic hypoxia
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Hypertension and cardiac dysfunction in male mice deficient in the alpha 1 subunit of soluble guanylate cyclase
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Gender-specific modulation of baseline vascular function and the response to arterial injury by soluble guanylate cyclase
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Soluble guanylate cyclase deficiency selectively abolishes NO-mediated pulmonary vasodilation and increases the pulmonary vascular remodeling response to chronic hypoxia
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Soluble guanylate cyclase deficiency selectively abolishes NO-mediated pulmonary vasodilation and increases the pulmonary vascular remodeling response to chronic hypoxia