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Extracellular ATP and P₂X₇ receptor exert context-specific immunogenic effects after immunogenic cancer cell death
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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)
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- Journal Article
- A1
- open access
BNIP3 supports melanoma cell migration and vasculogenic mimicry by orchestrating the actin cytoskeleton
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Genetic association and functional role of Crohn disease risk alleles involved in microbial sensing, autophagy, and endoplasmic reticulum (ER) stress
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Guidelines for the use and interpretation of assays for monitoring autophagy
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18th Euroconference on Apoptosis in Ghent, Belgium, 1-4 September 2010
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Immunogenic cell death, DAMPs and anticancer therapeutics: An emerging amalgamation
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Caspase-mediated cleavage of Beclin-1 inactivates Beclin-1-induced autophagy and enhances apoptosis by promoting the release of proapoptotic factors from mitochondria
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Blocking tumor cell eicosanoid synthesis by GPx4 impedes tumor growth and malignancy
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Role of endoplasmic reticulum depletion and multidomain proapoptotic BAX and BAK proteins in shaping cell death after hypericin-mediated photodynamic therapy
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Tumor cell eicosanoids promote tumor growth, metastasis and escape from angiodestructive tumor therapy
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Activation of p38 MAPK is required for Bax translocation to mitochondria, cytochrome c release and apoptosis induced by UVB irradiation in human keratinocytes
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Ultraviolet B radiation-induced apoptosis in human keratinocytes: cytosolic activation of procaspase-8 and the role of Bcl-2
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Different pathways mediate cytochrome c release after photodynamic therapy with hypericin.
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p38 mitogen-activated protein kinase regulates a novel, caspase-independent pathway for the mitochondrial cytochrome c release in ultraviolet B radiation-induced apoptosis.
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The activation of the c-Jun N-terminal kinase and p38 mitogen-activated protein kinase signaling pathways protects HeLa cells from apoptosis following photodynamic therapy with hypericin.
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Hypericin-induced photosensitization of HeLa cells leads to apoptosis or necrosis - Involvement of cytochrome c and procaspase-3 activation in the mechanism of apoptosis.
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Identification of two Physarum casein kinase II enzymes that phosphorylate fragmin, an endogenous actin-binding protein.
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Casein kinase-1 phosphorylates the p75 tumor-necrosis-factor receptor and negatively regulates tumor-necrosis-factor signaling for apoptosis.
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TUMOR-NECROSIS-FACTOR STIMULATES MULTIPLE SERINE THREONINE PROTEIN-KINASES IN SWISS 3T3 AND L929 CELLS - IMPLICATION OF CASEIN KINASE-2 AND EXTRACELLULAR SIGNAL-REGULATED KINASES IN THE TUMOR-NECROSIS-FACTOR SIGNAL TRANSDUCTION PATHWAY.