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Induction and repression of NF-kappaB-driven inflammatory genes.
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Regulation of the transcriptional activity of the nuclear factor-kappa B p65 subunit.
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Glucocorticoid repression of AP-1 is not mediated by competition for nuclear coactivators.
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Signal transduction by tumor necrosis factor and gene regulation of the inflammatory cytokine interleukin-6.
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In search of the molecular mechanism of gene repression by glucocorticoids
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In search of the molecular mechanism of gene repression by glucocorticoids.
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- Journal Article
- A1
- open access
The zinc finger protein A20 inhibits TNF-induced NF-kappa B-dependent gene expression by interfering with an RIP- or TRAF2-mediated transactivation signal and directly binds to a novel NF-kappa B-inhibiting protein ABIN.
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Dissociated glucocorticoids with anti-inflammatory potential repress interleukin-6 gene expression by a nuclear factor-kappa B-dependent mechanism.
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N-acetyl-L-cysteine inhibits primary human T cell responses at the dendritic cell level: Association with NF-kappa B inhibition.
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Peroxisome proliferator-activated receptor alpha negatively regulates the vascular inflammatory gene response by negative cross-talk with transcription factors NF-kappa B and AP-1.