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Serum vitamin C concentration is low in peripheral arterial disease and is associated with inflammation and severity of atherosclerosis (with comment: Vitamin C depletion linked to inflammation in peripheral artery disease. M. Larkin Lancet, 357, 2001, 1184).
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Haptoglobin polymorphism and peripheral arterial occlusive disease
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Is angiotensin-converting enzyme I/D polymorphism involved in cardiovascular complications in essential hypertension ?
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Vitamin D binding protein polymorphism and polymetabolic syndrome in essential hypertension
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Haptoglobin polymorphism and hypertension in the elderly
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Angiotensin-I converting enzyme (ACE) gene polymorphism and the erythrocyte sodium-lithium countertransport in hypertension.
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Genetic polymorphisms and erythrocyte sodium-lithium countertransport in essential hypertension
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Refractory hypertension is associated with haptoglobin 2-2 phenotype
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Sodium-lithium countertransport and vitamin D binding protein polymorphism in essential arterial hypertension.
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MN blood-group, a genetic-marker for essential arterial-hypertension in young-adults.
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MN blood group phenotype, a genetic risk factor for early onset of essential arterial hypertension
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MN blood group, a genetic marker for essential arterial hypertension
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Coagulation system activation and increase of D-dimers levels in peripheral arterial occlusive disease
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MN bloodgroup, a genetic marker for arterial pressure height and body mass index in treated essential hypertension
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Haptoglobin polymorphism and peripheral arterial occlusive disease
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MN blood group, a genetic marker for essential arterial hypertension.
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MN blood group, a genetic marker for arterial pressure height and body mass index in treated essential hypertension
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MN blood group, a genetic marker for arterial blood pressure height and body mass index in treated essential hypertension.
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Flow cytometric detection of platelet response to ADP in peripheral arterial occlusive disease after treadmill exercise
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Calculation of acute myocardial infarction size making use of cumulative myoglobin release in serum
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Increased plasma-levels of molecular markers of coagulation activation and secondary fibrinolysis in peripheral arterial occlusive disease
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HAPTOGLOBIN POLYMORPHISM AND COMPLICATIONS IN ESTABLISHED ESSENTIAL ARTERIAL-HYPERTENSION.
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Prothrombin activation fragment 1+2, thrombin-antithrombin III complex and D-dimer determinations in peripheral arterial occlusive disease - influence of treadmill exercise.
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Haptoglobin 2-2, a genetic marker for therapy resistance in established essential hypertension
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The relation between haptoglobin polymorphism and therapeutic needs in essential arterial hypertension.
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Haptoglobin 2-2, a genetic marker for therapy resistance in established essential hypertension. Eur. Heart J., 1992, 13 (Abstract Supplement), p. 85.
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Haptoglobin phenotype 2-2: agenetic marker for therapy - resistant essential hypertension
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Basal fibrinolysis in essential hypertension and peripheral arterial disease
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Activation of the clotting cascade in peripheral arterial occlusive disease. Abstract of the 12th International Congress on Thrombosis
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Fibrinolytische parameters tijdens coronaire angioplastiek
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Haptoglobin 2-2, a genetic marker for therapy resistance in established essential hypertension
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Acute myocardial infarction size and myoglobin release into serum.
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Monitoring of haptoglobin concentration during the early phase of acute myocardial infarction.
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Haptoglobin 2-2, a genetic marker for therapy resistance in established essential hypertension
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A new method for calculation of infarct size making use of cumulative myoglobin release.
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Can creatine predict further major cardiovascular events after acute myocardial infarction ?
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Low serum creatine kinase in patients with infective endocarditis
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Basal fibrinolysis in essential-hypertension and peripheral arterial disease