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GSDMD drives canonical inflammasome-induced neutrophil pyroptosis and is dispensable for NETosis
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- Journal Article
- A1
- open access
Bacillus anthracis induces NLRP3 inflammasome activation and caspase-8-mediated apoptosis of macrophages to promote lethal anthrax
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- Journal Article
- A1
- open access
MCC950/CRID3 potently targets the NACHT domain of wild-type NLRP3 but not disease-associated mutants for inflammasome inhibition
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Structure of S-layer protein Sap reveals a mechanism for therapeutic intervention in anthrax
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- Journal Article
- A1
- open access
Caspase-1 engagement and TLR-induced c-FLIP expression suppress ASC/caspase-8-dependent apoptosis by inflammasome sensors NLRP1b and NLRC4
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Nlrp6-and ASC-dependent inflammasomes do not shape the commensal gut microbiota composition
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Familial Mediterranean fever mutations lift the obligatory requirement for microtubules in Pyrin inflammasome activation
(2016) PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA. 113(50). p.14384-14389 -
The NEK-sus of the NLRP3 inflammasome
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Does caspase-12 suppress inflammasome activation?
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TNFR1-induced lethal inflammation is mediated by goblet and Paneth cell dysfunction
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Generation and characterization of small single domain antibodies inhibiting human tumor necrosis factor receptor 1
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Determining differentially expressed miRNAs and validating miRNA-target relationships using the SPRET/Ei mouse strain
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Antisense oligonucleotides against TNFR1 prevent toxicity of TNF/IFNγ treatment in mouse tumor models
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NOD-like receptors interfacing the immune and reproductive systems
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Necrostatin-1 analogues: critical issues on the specificity, activity and in vivo use in experimental disease models
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Matrix metalloprotease 8-dependent extracellular matrix cleavage at the blood-CSF barrier contributes to lethality during systemic inflammatory diseases
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RIP Kinase-dependent necrosis drives lethal systemic inflammatory response syndrome
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Tumor necrosis factor Inhibits glucocorticoid receptor function in mice a strong signal toward lethal shock
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Strategies to inhibit the toxicity of systemic TNF treatment
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Type I interferon drives tumor necrosis factor-induced lethal shock