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Segmental chromosomal alterations lead to a higher risk of relapse in infants with MYCN-non-amplified localised unresectable/disseminated neuroblastoma (a SIOPEN collaborative study)
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Risk estimation of neuroblastoma patients by gene expression based classification
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Quality assessment of genetic markers used for therapy stratification
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Comparative genomic hybridization (CGH) analysis of stage 4 neuroblastoma reveals high frequency of 11Q deletion in tumors lacking MYCN amplification.
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Deletion of chromosome 11q is the most frequent deletion detected by CGH in metastatic neuroblastoma lacking MYCN amplification