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Assisted oocyte activation does not overcome recurrent embryo developmental problems
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Vitrification negatively affects the Ca2+-releasing and activation potential of mouse oocytes, but vitrified oocytes are potentially useful for diagnostic purposes
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- Journal Article
- A1
- open access
Diagnosis and treatment of male infertility-related fertilization failure
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Comparative study of preimplantation development following distinct assisted oocyte activation protocols in a PLC-zeta knockout mouse model
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Assisted oocyte activation significantly increases fertilization and pregnancy outcome in patients with low and total failed fertilization after intracytoplasmic sperm injection : a 17-year retrospective study
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Assessment of the calcium releasing machinery in oocytes that failed to fertilize after conventional ICSI and assisted oocyte activation
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- Journal Article
- A1
- open access
Strontium fails to induce Ca2+ release and activation in human oocytes despite the presence of functional TRPV3 channels
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Culture conditions affect Ca2+ release in artificially activated mouse and human oocytes
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Human oocyte calcium analysis predicts the response to assisted oocyte activation in patients experiencing fertilization failure after ICSI
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Single Ca2+ transients vs oscillatory Ca2+ signaling for assisted oocyte activation : limitations and benefits
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- Journal Article
- A1
- open access
PLCζ is the physiological trigger of the Ca2+ oscillations that induce embryogenesis in mammals but conception can occur in its absence
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PLCζ disruption with complete fertilization failure in normozoospermia
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Neonatal and neurodevelopmental outcome of children aged 3-10 years born following assisted oocyte activation
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Language development of children born following intracytoplasmic sperm injection (ICSI) combined with assisted oocyte activation (AOA)
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Assisted oocyte activation following ICSI fertilization failure
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Diagnostic and prognostic value of calcium oscillatory pattern analysis for patients with ICSI fertilization failure
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Comparison of pre- and post-implantation development following the application of three artificial activating stimuli in a mouse model with round-headed sperm cells deficient for oocyte activation
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Globozoospermia is mainly due to DPY19L2 deletion via non-allelic homologous recombination involving two recombination hotspots