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Enhancement of polymethacrylate-mediated gene delivery by penetratin. (Lecture by P. Dubruel)
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Penetratin-membrane association: W48/R52/W56 shield the peptide from the aqueous phase
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Regulation of the ABCA1 transporter activity by protein kinase CK2
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Buffering properties of cationic polymethacrylates are not the only key to successful gene delivery
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Membrane interaction and cellular internalization of penetratin peptides
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Expression and activity of the nucleotide-binding domains of the human ABCA1 transporter
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Phosphorylation by protein kinase CK2 modulates the activity of the ATP binding cassette A1 transporter
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Structure and regulation of the ABCA transporters
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Cationic polymethacrylates as vectors for gene delivery: synthesis, physico-chemical and biological evaluation (Poster by P. Dubruel)
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Cationic polymethacrylates as vectors for gene delivery: synthesis, physico-chemical and biological evaluation (Oral Communication by P. Dubruel)
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Synthetic polyamines as vectors for gene delivery: structure-property relation
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Physicochemical and biological evaluation of cationic polymethacrylates as vectors for gene delivery
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Poly-L-glutamic acid derivatives as multifunctional vectors for gene delivery. Part B. Biological evaluation (vol 4, pg 1177, 2003)
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Poly-L-glutamic acid derivatives as multifunctional vectors for gene delivery. Part B. Biological evaluation
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Characterization of the ABCA transporter subfamily: Identification of prokaryotic and eukaryotic members, phylogeny and topology
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Activation of lecithin cholesterol acyl transferase (LCAT) by apolipoproteins
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Interaction of nanostructured polyelectrolyte complexes with cells
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Interaction of nanostructured polyelectrolyte complexes with cells (Poster by P. Dubruel)
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Tryptophan fluorescence study of the interaction of penetratin peptides with model membranes.
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Cationic polymethacrylates as vectors for gene therapy
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Specific docking of apolipoprotein A-I at the cell surface requires a functional ABCA1 transporter.
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Comparison of the aggregation properties, secondary structure and apoptotic effects of wild-type, Flemish and Dutch N-terminally truncated amyloid beta peptides.
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A hydrophobic cluster at the surface of the human plasma phospholipid transfer protein is critical for activity on high density lipoproteins.
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ApoE protects cortical neurones against neurotoxicity induced by the non-fibrillar C-terminal domain of the amyloid-beta peptide.
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Effect of mutations of N- and C-terminal charged residues on the activity of LCAT.
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Three arginine residues in apolipoprotein A-I are critical for activation of lecithin: cholesterol acyltransferase.
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Structure and function of lecithin cholesterol acyl transferase: new insights from structural predictions and animal models.
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Apoptosis induced in neuronal cells by C-terminal amyloid beta-fragments is correlated with their aggregation properties in phospholipid membranes.
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Characterization of recombinant wild type and site-directed mutations of apolipoprotein C-III: Lipid binding, displacement of ApoE, and inhibition of lipoprotein lipase.
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A mechanism of membrane neutral lipid acquisition by the microsomal triglyceride transfer protein.
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Relationship between structure and biochemical phenotype of lecithin: cholesterol acyltransferase (LCAT) mutants causing fish-eye disease.
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Characterization of the lipid-binding properties and lipoprotein lipase inhibition of a novel apolipoprotein C-III variant Ala23Thr.
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Neurotoxicity of the putative transmembrane domain of the prion protein.
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Characterization and functional studies of lipoproteins, lipid transfer proteins, and lecithin: cholesterol acyltransferase in CSF of normal individuals and patients with Alzheimer's disease.
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Headgroup specificity of lecithin cholesterol acyltransferase for monomeric and vesicular phospholipids.
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Lipids, apolipoproteins and LCAT in cerebrospinal fluid of normal individuals and patients with Alzhelmer's disease
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Characterisation of functional residues in the interfacial recognition domain of lecithin cholesterol acyltransferase.
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Molecular determinants of the interaction between the C-terminal domain of Alzheimer's beta-amyloid peptide and apolipoprotein E alpha-helices.
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Contribution of the hydrophobicity gradient to the secondary structure and activity of fusogenic peptides.
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LCAT defects and low HDL levels.
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Effects of natural mutations in lecithin:cholesterol acyltransferase on the enzyme structure and activity
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Baculovirus expression and biochemical characterization of the human microsomal triglyceride transfer protein.
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The nonfibrillar amyloid beta-peptide induces apoptotic neuronal cell death: Involvement of its C-terminal fusogenic domain.
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beta-amyloid peptide interacts specifically with the carboxy-terminal domain of human apolipoprotein E: Relevance to Alzheimer's disease.
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Abnormal capacity to induce cholesterol efflux and a new LpA-I pre-beta particle in type 2 diabetic patients.
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Effect of apo E phenotype on plasma postprandial triglyceride levels in young male adults with and without a familial history of myocardial infarction: the EARS II study.
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Characterization of functional residues in the interfacial recognition domain of lecithin cholesterol acyltransferase (LCAT).
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The structure of vitellogenin provides a molecular model for the assembly and secretion of atherogenic lipoproteins.
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The effect of age and lifestyle factors on plasma levels of apolipoprotein E plasma levels
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The effect of lifestyle factors on apolipoprotein E levels
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Comparison of three fatty meals in healthy normolipidaemic men: high post-prandial retinyl ester response to soybean oil.
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Apolipoprotein localization in reconstituted HDL particles: fluorescence energy transfer study.
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Enhanced efficiency of a targeted fusogenic peptide.
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Contribution of apo A II and LCAT oblique peptides to HDL metabolism
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Contribution of apo AII and LCAT oblique peptides to HDL metabolism
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Decreased postprandial high density lipoprotein cholesterol and apolipoproteins A-I and E in normolipidemic smoking men: relations with lipid transfer proteins and LCAT activities.
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Contribution of the hydrophobicity gradient of an amphipathic peptide to its mode of association with lipids.
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Feasibility of a recombinant human apolipoprotein E reference material.
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Displacement of apo A-I from HDL by apo A-II or its C-terminal helix promotes the formation of pre-beta(1) migrating particles and decreases LCAT activation.
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The C-terminal helix of human apolipoprotein AII promotes the fusion of unilamellar liposomes and displaces apolipoprotein AI from high-density lipoproteins.
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Branched synthetic peptide constructs mimic cellular binding and efflux of apolipoprotein AI in reconstituted high density lipoproteins.
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A proposed architecture for lecithin cholesterol acyl transferase (LCAT): Identification of the catalytic triad and molecular modeling.
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Apolipoprotein E inhibits liposome fusion induced by the Alzheimer's beta-amyloid peptide.
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Structural organization of lipid phase and protein-lipid interface in apolipoprotein-phospholipid recombinants: Influence of cholesterol.
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Variation at the lipoprotein lipase and apolipoprotein AI-CIII gene loci are associated with fasting lipid and lipoprotein traits in a population sample from Iceland: Interaction between genotype, gender, and smoking status.
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Peptides in membranes: Tipping the balance of membrane stability.
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Design of a new class of amphipathic helical peptides for the plasma apolipoproteins that promote cellular cholesterol efflux but do not activate LCAT.
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Composition and structural and functional properties of discoidal and spherical phospholipid-ApoE3 complexes.
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Interactions between lifestyle-related factors and the ApoE polymorphism on plasma lipids and apolipoproteins - The EARS study.
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Specific modulation of the fusogenic properties of the Alzheimer beta-amyloid peptide by apolipoprotein E isoforms.
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Determinants of postprandial lipemia in men with coronary artery disease and low levels of HDL cholesterol.
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Structural and functional properties of the 154-171 wild-type and variant peptides of human lecithin-cholesterol acyltransferase.
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The 118-135 peptide lot the human prion protein forms amyloid fibrils and induces liposome fusion.
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Determinants of lipoprotein(a) levels in a middle-aged working population.
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Apolipoprotein E polymorphism in middle-aged Belgian men: Phenotype distribution and relation to serum lipids and lipoproteins.
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Lipoprotein [a].
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Dual effects of oxidized low-density lipoprotein on immune-stimulated nitric oxide and prostaglandin release in macrophages.
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Single-step purification of two functional human apolipoprotein E variants hyperexpressed in Escherichia coli.
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Branched synthetic constructs that mimic the physico-chemical properties of apolipoprotein AI in reconstituted high-density lipoproteins.
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Fusogenic properties of the C-terminal domain of the Alzheimer beta-amyloid peptide.
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Lipid-binding properties of synthetic peptide fragments of human apolipoprotein A-II.
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PHYSIOLOGICAL SIGNIFICANCE OF APOLIPOPROTEIN MUTANTS.
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COMPARISON OF LIPID-BINDING AND LECITHIN-CHOLESTEROL ACYLTRANSFERASE ACTIVATION OF THE AMINO-TERMINAL AND CARBOXYL-TERMINAL DOMAINS OF HUMAN APOLIPOPROTEIN E3.
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RELATION OF FIBRINOGEN TO LIFE-STYLES AND TO CARDIOVASCULAR RISK-FACTORS IN A WORKING POPULATION.
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HELIX HELIX INTERACTIONS IN RECONSTITUTED HIGH-DENSITY-LIPOPROTEINS.
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ASSOCIATION OF SYNTHETIC PEPTIDE-FRAGMENTS OF HUMAN APOLIPOPROTEIN-A-I WITH PHOSPHOLIPIDS.
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Biomarkers of dietary fat composition in young adults with a parental history of premature coronary heart disease compared with controls. The EARS study.
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Plasma apolipoprotein concentrations in young adults with a parental history of premature coronary heart-disease and in control subjects - the EARS Study.
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A novel type of acquired form of hypolipoproteinemia
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POLYMORPHISMS IN THE APOLIPOPROTEIN (APO) AI-CIII-AIV GENE-CLUSTER - DETECTION OF GENETIC-VARIATION DETERMINING PLASMA APO-AI, APO-CIII AND APO-AIV CONCENTRATIONS.
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Acquired Hypolipoproteinemia.
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PLASMA LIPOPROTEIN(A) VALUES AND SEVERITY OF CORONARY-ARTERY DISEASE IN A LARGE POPULATION OF PATIENTS UNDERGOING CORONARY ANGIOGRAPHY.
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Determinanten van het vetverbruik in een algemene populatie
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An unusual case of acquired hypolipemia and LCAT deficiency. Proc. 9th Intern. Symposium on Atherosclerosis, Chicago, 1991, p. 100.
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An unusual case of acquired hypolipemia and LCAT deficiency. Proc. 57th European Atherosclerosis Society Congress, Lissabon 1991, p. 90.
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An unusual case of acquired hypolipaemia and LCAT deficiency. Molecular Biology of Atherosclerosis, P-oceedings of the 57th European Atherosclerosis Society Meeting, Ed. M.J. Halpern, John Libbey & Cie Ltd, Chapter 28, 1992, pp. 137-139.