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Intermediate domain of receptor-interacting protein kinase 1 (RIPK1) determines switch between necroptosis and RIPK1 kinase-dependent apoptosis
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The mitochondrial serine protease HtrA2/Omi cleaves RIP1 during apoptosis of Ba/F3 cells induced by growth factor withdrawal
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Necroptosis, necrosis and secondary necrosis converge on similar cellular disintegration features
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Necrosis: molecular mechanisms and physiological roles
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Molecular mechanisms and pathophysiology of necrotic cell death
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Caspases in cell survival, proliferation and differentiation
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RIP1, a kinase on the crossroads of a cell's decision to live or die
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Caspase inhibitors promote alternative cell death pathways
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Necrosis, a well-orchestrated form of cell demise: Signalling cascades, important mediators and concomitant immune response
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Fas induced necrosis.