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A bispecific Clec9A-PD-L1 targeted type I interferon profoundly reshapes the tumor microenvironment towards an antitumor state
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ZBTB32 performs crosstalk with the glucocorticoid receptor and is crucial in glucocorticoid responses to starvation
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Specific targeting of IL-1β activity to CD8+ T cells allows for safe use as a vaccine adjuvant
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Selected IL-1 activity on CD8+ T cells allows for safe enhancement of cellular immune responses
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Safe eradication of large established tumors using neovasculature-targeted tumor necrosis factor-based therapies
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Towards the use of interleukin-1β as vaccine adjuvant
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Mechanisms underlying the functional cooperation between PPARα and GRα to attenuate inflammatory responses
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Strategies and compounds to circumvent glucocorticoid-induced side effects
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The autophagy receptor SQSTM1/p62 mediates anti-inflammatory actions of the selective NR3C1/glucocorticoid receptor modulator compound A (CpdA) in macrophages
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Co-activation of glucocorticoid receptor and peroxisome proliferator-activated receptor-γ in murine skin prevents worsening of atopic march
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Treatment with mRNA coding for the necroptosis mediator MLKL induces antitumor immunity directed against neo-epitopes
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Glucocorticoid receptor-mediated transactivation is hampered by Striatin-3, a novel interaction partner of the receptor
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Structure and antagonism of the receptor complex mediated by human TSLP in allergy and asthma
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The interactome of the glucocorticoid receptor and its influence on the actions of glucocorticoids in combatting inflammatory and infectious diseases
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Modulation of protein-protein interactions for the development of novel therapeutics
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Chromatin recruitment of activated AMPK drives fasting response genes co-controlled by GR and PPARα
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Proteome-scale binary interactomics in human cells
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Leptin's metabolic and immune functions can be uncoupled at the ligand/receptor interaction level
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Genome dynamics of the human embryonic kidney 293 lineage in response to cell biology manipulations
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Selective modulation of the glucocorticoid receptor can distinguish between transrepression of NF-κB and AP-1