Ghent University Academic Bibliography

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Synaptogyrin-3 mediates presynaptic dysfunction induced by Tau

Joseph McInnes, Keimpe Wierda, An Snellinx, Laura Bounti, Yu-Chun Wang, Ilie-Cosmin Stancu, Nuno Apóstolo, Kris Gevaert (UGent) , Ilse Dewachter, Tara L Spires-Jones, et al.

(2018) NEURON. 97(4). p.823-835
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- Book Chapter
Neuropathobiology in transgenic mice : the case of Alzheimer's disease

Jo Van Dorpe (UGent) , Kurt Spittaels, Chris Van den Haute, Ilse Dewachter, Dieder Moerchars, Hugo Geerts and Fred Van Leuven

(2003) Transgenic mouse : methods and protocols. In Methods in Molecular Biology 209. p.333-361
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Reduction of amyloid load and cerebral damage in a transgenic mouse model of Alzheimer's disease by treatment with a β-sheet breaker peptide

Bruno Permanne, Céline Adessi, Gabriela P Saborio, Santiago Fraga, Marie-José Frossard, Jo Van Dorpe (UGent) , Ilse Dewachter, William A Banks, Fred Van Leuven and Claudio Soto

(2002) FASEB JOURNAL. 16(6). p.860-862
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Modelling Alzheimer's disease in multiple transgenic mice

Ilse Dewachter, Dieder Moechars, Jo Van Dorpe (UGent) , Ina Tesseur, Chris Van den Haute, Kurt Spittaels and Fred Van Leuven

(2001) Biochemical Society Symposium. 67. p.203-210
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- Journal Article
- A1
- open access
Aging increased amyloid peptide and caused amyloid plaques in brain of old APP/V717I transgenic mice by a different mechanism than mutant presenilin1

Ilse Dewachter, Jo Van Dorpe (UGent) , Liesbet Smeijers, Martine Gilis, Cuno Kuipéri, Isabelle Laenen, Nathalie Caluwaerts, Dieder Moechars, Frédéric Checler, Hugo Vanderstichele, et al.

(2000) JOURNAL OF NEUROSCIENCE. 20(17). p.6452-6458
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Prominent cerebral amyloid angiopathy in transgenic mice overexpressing the London mutant of human APP in neurons

Jo Van Dorpe (UGent) , Liesbet Smeijers, Ilse Dewachter, Dieter Nuyens, Kurt Spittaels, Chris Van den Haute, Marc Mercken, Dieder Moechars, Isabelle Laenen, Cuno Kuiperi, et al.

(2000) AMERICAN JOURNAL OF PATHOLOGY. 157(4). p.1283-1298

Academic Bibliography

Synaptogyrin-3 mediates presynaptic dysfunction induced by Tau

Neuropathobiology in transgenic mice : the case of Alzheimer's disease

Reduction of amyloid load and cerebral damage in a transgenic mouse model of Alzheimer's disease by treatment with a β-sheet breaker peptide

Modelling Alzheimer's disease in multiple transgenic mice

Aging increased amyloid peptide and caused amyloid plaques in brain of old APP/V717I transgenic mice by a different mechanism than mutant presenilin1

Prominent cerebral amyloid angiopathy in transgenic mice overexpressing the London mutant of human APP in neurons

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