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Regulation of the expression and processing of caspase-12
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A matrix-assisted laser desorption ionization post-source decay (MALDI-PSD) analysis of proteins released from isolated liver mitochondria treated with recombinant truncated Bid
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Signaling to gene activation and cell death by tumor necrosis factor receptors and Fas
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Tipping the balance between necrosis and apoptosis in human and murine cells treated with interferon and dsRNA
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Caspases are not localized in mitochondria during life or death
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The role of mitochondrial factors in apoptosis: a Russian roulette with more than one bullet.
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TNF-alpha receptors simultaneously activate Ca2+ mobilisation and stress kinases in cultured sensory neurones.
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The p55 tumour necrosis factor receptor TNFR1 contains a trans-Golgi network localization signal in the C-terminal region of its cytoplasmic tail.
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Head involution defective (Hid)-triggered apoptosis requires caspase-8 but not FADD (Fas-associated death domain) and is regulated by Erk in mammalian cells.
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The serine protease Omi/HtrA2 is released from mitochondria during apoptosis. Omi interacts with caspase-inhibitor XIAP and induces enhanced caspase activity.