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Rbm7 in structural cells : a NEAT way to control fibrosis

Hamida Hammad (UGent) and Bart Lambrecht (UGent)
(2020) IMMUNITY. 52(3). p.429-431
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Abstract
The initial molecular events and the cell type(s) responsible for the development of fibrosis are unclear. Fukushima, Satoh, et al. find that increased expression of the nuclear exosome targeting complex component Rbm7 in lung epithelial cells promotes the degradation of the long non-coding RNA NEAT1, impairs DNA repair, and triggers apoptosis. Dying epithelial cells release chemokines that recruit atypical monocytes, which drive tissue fibrosis.

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Please use this url to cite or link to this publication:

MLA
Hammad, Hamida, and Bart Lambrecht. “Rbm7 in Structural Cells : A NEAT Way to Control Fibrosis.” IMMUNITY, vol. 52, no. 3, 2020, pp. 429–31, doi:10.1016/j.immuni.2020.02.008.
APA
Hammad, H., & Lambrecht, B. (2020). Rbm7 in structural cells : a NEAT way to control fibrosis. https://doi.org/10.1016/j.immuni.2020.02.008
Chicago author-date
Hammad, Hamida, and Bart Lambrecht. 2020. “Rbm7 in Structural Cells : A NEAT Way to Control Fibrosis.” IMMUNITY. https://doi.org/10.1016/j.immuni.2020.02.008.
Chicago author-date (all authors)
Hammad, Hamida, and Bart Lambrecht. 2020. “Rbm7 in Structural Cells : A NEAT Way to Control Fibrosis.” IMMUNITY. doi:10.1016/j.immuni.2020.02.008.
Vancouver
1.
Hammad H, Lambrecht B. Rbm7 in structural cells : a NEAT way to control fibrosis. Vol. 52, IMMUNITY. 2020. p. 429–31.
IEEE
[1]
H. Hammad and B. Lambrecht, “Rbm7 in structural cells : a NEAT way to control fibrosis,” IMMUNITY, vol. 52, no. 3. pp. 429–431, 2020.
@misc{8663007,
  abstract     = {{The initial molecular events and the cell type(s) responsible for the development of fibrosis are unclear. Fukushima, Satoh, et al. find that increased expression of the nuclear exosome targeting complex component Rbm7 in lung epithelial cells promotes the degradation of the long non-coding RNA NEAT1, impairs DNA repair, and triggers apoptosis. Dying epithelial cells release chemokines that recruit atypical monocytes, which drive tissue fibrosis.}},
  author       = {{Hammad, Hamida and Lambrecht, Bart}},
  issn         = {{1074-7613}},
  language     = {{eng}},
  number       = {{3}},
  pages        = {{429--431}},
  series       = {{IMMUNITY}},
  title        = {{Rbm7 in structural cells : a NEAT way to control fibrosis}},
  url          = {{http://dx.doi.org/10.1016/j.immuni.2020.02.008}},
  volume       = {{52}},
  year         = {{2020}},
}

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