Advanced search

Non-classical ProIL-1beta activation during mammary gland infection is pathogen-dependent but caspase-1 independent

Author
Organization

Citation

Please use this url to cite or link to this publication:

Chicago
Breyne, Koen, Steven Cool, Dieter Demon, Kristel Demeyere, Tom Vandenberghe, Peter Vandenabeele, Harald Carlsen, Wim Van Den Broeck, Niek Sanders, and Evelyne Meyer. 2014. “Non-classical ProIL-1beta Activation During Mammary Gland Infection Is Pathogen-dependent but Caspase-1 Independent.” In ICDI Symposium, Abstracts.
APA
Breyne, K., Cool, S., Demon, D., Demeyere, K., Vandenberghe, T., Vandenabeele, P., Carlsen, H., et al. (2014). Non-classical ProIL-1beta activation during mammary gland infection is pathogen-dependent but caspase-1 independent. ICDI Symposium, Abstracts. Presented at the International symposium on Mechanisms of Innate Immunity, Cell Death and Inflammation (ICDI symposium) 2014.
Vancouver
1.
Breyne K, Cool S, Demon D, Demeyere K, Vandenberghe T, Vandenabeele P, et al. Non-classical ProIL-1beta activation during mammary gland infection is pathogen-dependent but caspase-1 independent. ICDI Symposium, Abstracts. 2014.
MLA
Breyne, Koen, Steven Cool, Dieter Demon, et al. “Non-classical ProIL-1beta Activation During Mammary Gland Infection Is Pathogen-dependent but Caspase-1 Independent.” ICDI Symposium, Abstracts. 2014. Print.
@inproceedings{5721655,
  author       = {Breyne, Koen and Cool, Steven and Demon, Dieter and Demeyere, Kristel and Vandenberghe, Tom and Vandenabeele, Peter and Carlsen, Harald and Van Den Broeck, Wim and Sanders, Niek and Meyer, Evelyne},
  booktitle    = {ICDI Symposium, Abstracts},
  language     = {eng},
  location     = {Ghent, Belgium},
  title        = {Non-classical ProIL-1beta activation during mammary gland infection is pathogen-dependent but caspase-1 independent},
  year         = {2014},
}