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The Glu318Gly substitution in presenilin 1 is not causally related to Alzheimer disease

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VASCULAR DEMENTIA, MISSENSE MUTATIONS, GENE, ROTTERDAM

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Chicago
Dermaut, Bart, Marc Cruts, Arjen JC Slooter, Sofie Van Gestel, Chris De Jonghe, Hugo Vanderstichele, Eugeen Vanmechelen, et al. 1999. “The Glu318Gly Substitution in Presenilin 1 Is Not Causally Related to Alzheimer Disease.” American Journal of Human Genetics 64 (1): 290–292.
APA
Dermaut, B., Cruts, M., Slooter, A. J., Van Gestel, S., De Jonghe, C., Vanderstichele, H., Vanmechelen, E., et al. (1999). The Glu318Gly substitution in presenilin 1 is not causally related to Alzheimer disease. AMERICAN JOURNAL OF HUMAN GENETICS, 64(1), 290–292.
Vancouver
1.
Dermaut B, Cruts M, Slooter AJ, Van Gestel S, De Jonghe C, Vanderstichele H, et al. The Glu318Gly substitution in presenilin 1 is not causally related to Alzheimer disease. AMERICAN JOURNAL OF HUMAN GENETICS. 1999;64(1):290–2.
MLA
Dermaut, Bart, Marc Cruts, Arjen JC Slooter, et al. “The Glu318Gly Substitution in Presenilin 1 Is Not Causally Related to Alzheimer Disease.” AMERICAN JOURNAL OF HUMAN GENETICS 64.1 (1999): 290–292. Print.
@article{3201216,
  author       = {Dermaut, Bart and Cruts, Marc and Slooter, Arjen JC and Van Gestel, Sofie and De Jonghe, Chris and Vanderstichele, Hugo and Vanmechelen, Eugeen and Breteler, Monique M and Hofman, Albert and van Duijn, Cornelia M and Van Broeckhoven, Christine},
  issn         = {0002-9297},
  journal      = {AMERICAN JOURNAL OF HUMAN GENETICS},
  keyword      = {VASCULAR DEMENTIA,MISSENSE MUTATIONS,GENE,ROTTERDAM},
  language     = {eng},
  number       = {1},
  pages        = {290--292},
  title        = {The Glu318Gly substitution in presenilin 1 is not causally related to Alzheimer disease},
  url          = {http://dx.doi.org/10.1086/302200},
  volume       = {64},
  year         = {1999},
}

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