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Possible association of nicastrin polymorphisms and Alzheimer disease in the Finnish population

(2004) NEUROLOGY. 63(1). p.173-175
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Abstract
The authors previously reported that genetic variation in the gene coding for nicastrin (NCSTN) modified risk for familial early-onset Alzheimer disease (AD) in a Dutch population-based sample. Risk was highest in patients without an APOE epsilon4 allele. Here, they evaluated if NCSTN polymorphisms increased risk of AD in the eastern Finnish population. A significant difference in one haplotype was observed in AD patients without the APOE epsilon4 allele.
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Citation

Please use this url to cite or link to this publication:

MLA
Helisalmi, S, Bart Dermaut, M Hiltunen, et al. “Possible Association of Nicastrin Polymorphisms and Alzheimer Disease in the Finnish Population.” NEUROLOGY 63.1 (2004): 173–175. Print.
APA
Helisalmi, S., Dermaut, B., Hiltunen, M., Mannermaa, A., Van den Broeck, M., Lehtovirta, M., Koivisto, A., et al. (2004). Possible association of nicastrin polymorphisms and Alzheimer disease in the Finnish population. NEUROLOGY, 63(1), 173–175.
Chicago author-date
Helisalmi, S, Bart Dermaut, M Hiltunen, A Mannermaa, M Van den Broeck, M Lehtovirta, AM Koivisto, et al. 2004. “Possible Association of Nicastrin Polymorphisms and Alzheimer Disease in the Finnish Population.” Neurology 63 (1): 173–175.
Chicago author-date (all authors)
Helisalmi, S, Bart Dermaut, M Hiltunen, A Mannermaa, M Van den Broeck, M Lehtovirta, AM Koivisto, S Iivonen, M Cruts, H Soininen, and C Van Broeckhoven. 2004. “Possible Association of Nicastrin Polymorphisms and Alzheimer Disease in the Finnish Population.” Neurology 63 (1): 173–175.
Vancouver
1.
Helisalmi S, Dermaut B, Hiltunen M, Mannermaa A, Van den Broeck M, Lehtovirta M, et al. Possible association of nicastrin polymorphisms and Alzheimer disease in the Finnish population. NEUROLOGY. 2004;63(1):173–5.
IEEE
[1]
S. Helisalmi et al., “Possible association of nicastrin polymorphisms and Alzheimer disease in the Finnish population,” NEUROLOGY, vol. 63, no. 1, pp. 173–175, 2004.
@article{3200644,
  abstract     = {The authors previously reported that genetic variation in the gene coding for nicastrin (NCSTN) modified risk for familial early-onset Alzheimer disease (AD) in a Dutch population-based sample. Risk was highest in patients without an APOE epsilon4 allele. Here, they evaluated if NCSTN polymorphisms increased risk of AD in the eastern Finnish population. A significant difference in one haplotype was observed in AD patients without the APOE epsilon4 allele.},
  author       = {Helisalmi, S and Dermaut, Bart and Hiltunen, M and Mannermaa, A and Van den Broeck, M and Lehtovirta, M and Koivisto, AM and Iivonen, S and Cruts, M and Soininen, H and Van Broeckhoven, C},
  issn         = {0028-3878},
  journal      = {NEUROLOGY},
  keywords     = {RISK},
  language     = {eng},
  number       = {1},
  pages        = {173--175},
  title        = {Possible association of nicastrin polymorphisms and Alzheimer disease in the Finnish population},
  url          = {http://dx.doi.org/10.1212/01.WNL.0000133153.98139.4E},
  volume       = {63},
  year         = {2004},
}

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