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Delayed neurological deterioration following acute carbon monoxyde poisoning : comparison of clinical outcome, neuro-imaging and positron emission tomography findings

(1991) CEREBROVASCULAR DISEASES. 1(5). p.273-280
Author
Organization
Abstract
In 8 patients with delayed neurological deterioration, following acute CO poisoning, the clinical outcome, the neuroimaging and the positron emission tomography (PET) findings were compared. The severity of the neuropsychiatric sequelae correlated well with the changes of blood flow and oxygen metabolism and the CT scan findings in the brain. Nuclear magnetic resonance (NMR) appeared to have less predictive value on the clinical outcome. In the severe cases of post-CO encephalopathy, the ischemic nature of the metabolic changes could be demonstrated in the cortical regions by PET. However, it could not be determined whether the disturbances in the basal ganglia and cerebral white matter were only due to ischemia or to an additional cytotoxic effect of CO.
Keywords
RESPONSES, COMPUTERIZED TOMOGRAPHY, HYPOXIA, LEUKOENCEPHALOPATHY, BROMOCRIPTINE, PARKINSONISM, DELAYED NEUROLOGICAL DETERIORATION, TOXICITY, CARBON MONOXIDE POISONING, POSITRON EMISSION TOMOGRAPHY, NUCLEAR MAGNETIC RESONANCE, CEREBRAL BLOOD-FLOW, SEQUELAE, INTOXICATION, BRAIN

Citation

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Chicago
De Reuck, Jacques, J Van Aken, D Decoo, H Petit, D Leys, Ignace Lemahieu, and Karel Strijckmans. 1991. “Delayed Neurological Deterioration Following Acute Carbon Monoxyde Poisoning : Comparison of Clinical Outcome, Neuro-imaging and Positron Emission Tomography Findings.” Cerebrovascular Diseases 1 (5): 273–280.
APA
De Reuck, J., Van Aken, J., Decoo, D., Petit, H., Leys, D., Lemahieu, I., & Strijckmans, K. (1991). Delayed neurological deterioration following acute carbon monoxyde poisoning : comparison of clinical outcome, neuro-imaging and positron emission tomography findings. CEREBROVASCULAR DISEASES, 1(5), 273–280.
Vancouver
1.
De Reuck J, Van Aken J, Decoo D, Petit H, Leys D, Lemahieu I, et al. Delayed neurological deterioration following acute carbon monoxyde poisoning : comparison of clinical outcome, neuro-imaging and positron emission tomography findings. CEREBROVASCULAR DISEASES. 1991;1(5):273–80.
MLA
De Reuck, Jacques, J Van Aken, D Decoo, et al. “Delayed Neurological Deterioration Following Acute Carbon Monoxyde Poisoning : Comparison of Clinical Outcome, Neuro-imaging and Positron Emission Tomography Findings.” CEREBROVASCULAR DISEASES 1.5 (1991): 273–280. Print.
@article{208072,
  abstract     = {In 8 patients with delayed neurological deterioration, following acute CO poisoning, the clinical outcome, the neuroimaging and the positron emission tomography (PET) findings were compared. The severity of the neuropsychiatric sequelae correlated well with the changes of blood flow and oxygen metabolism and the CT scan findings in the brain. Nuclear magnetic resonance (NMR) appeared to have less predictive value on the clinical outcome. In the severe cases of post-CO encephalopathy, the ischemic nature of the metabolic changes could be demonstrated in the cortical regions by PET. However, it could not be determined whether the disturbances in the basal ganglia and cerebral white matter were only due to ischemia or to an additional cytotoxic effect of CO.},
  author       = {De Reuck, Jacques and Van Aken, J and Decoo, D and Petit, H and Leys, D and Lemahieu, Ignace and Strijckmans, Karel},
  issn         = {1015-9770},
  journal      = {CEREBROVASCULAR DISEASES},
  keyword      = {RESPONSES,COMPUTERIZED TOMOGRAPHY,HYPOXIA,LEUKOENCEPHALOPATHY,BROMOCRIPTINE,PARKINSONISM,DELAYED NEUROLOGICAL DETERIORATION,TOXICITY,CARBON MONOXIDE POISONING,POSITRON EMISSION TOMOGRAPHY,NUCLEAR MAGNETIC RESONANCE,CEREBRAL BLOOD-FLOW,SEQUELAE,INTOXICATION,BRAIN},
  language     = {eng},
  number       = {5},
  pages        = {273--280},
  title        = {Delayed neurological deterioration following acute carbon monoxyde poisoning : comparison of clinical outcome, neuro-imaging and positron emission tomography findings},
  url          = {http://dx.doi.org/10.1159/000108854},
  volume       = {1},
  year         = {1991},
}

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