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The endothelial monocyte-activating polypeptide II (EMAP II) is a substrate for caspase-7.

HA BEHRENSDORF, Marc Van De Craen, UE KNIES, Peter Vandenabeele UGent and M CLAUSS (2000) FEBS LETTERS. 466(1). p.143-147
Please use this url to cite or link to this publication:
author
organization
year
type
journalArticle (original)
publication status
published
subject
journal title
FEBS LETTERS
FEBS Lett.
volume
466
issue
1
pages
143-147 pages
Web of Science type
Article
Web of Science id
000085022000030
ISSN
0014-5793
DOI
10.1016/S0014-5793(99)01777-9
language
English
UGent publication?
yes
classification
A1
id
125923
handle
http://hdl.handle.net/1854/LU-125923
date created
2004-01-14 13:36:00
date last changed
2016-12-19 15:38:51
@article{125923,
  author       = {BEHRENSDORF, HA and Van De Craen, Marc and KNIES, UE and Vandenabeele, Peter and CLAUSS, M},
  issn         = {0014-5793},
  journal      = {FEBS LETTERS},
  language     = {eng},
  number       = {1},
  pages        = {143--147},
  title        = {The endothelial monocyte-activating polypeptide II (EMAP II) is a substrate for caspase-7.},
  url          = {http://dx.doi.org/10.1016/S0014-5793(99)01777-9},
  volume       = {466},
  year         = {2000},
}

Chicago
BEHRENSDORF, HA, Marc Van De Craen, UE KNIES, Peter Vandenabeele, and M CLAUSS. 2000. “The Endothelial Monocyte-activating Polypeptide II (EMAP II) Is a Substrate for Caspase-7.” Febs Letters 466 (1): 143–147.
APA
BEHRENSDORF, H., Van De Craen, M., KNIES, U., Vandenabeele, P., & CLAUSS, M. (2000). The endothelial monocyte-activating polypeptide II (EMAP II) is a substrate for caspase-7. FEBS LETTERS, 466(1), 143–147.
Vancouver
1.
BEHRENSDORF H, Van De Craen M, KNIES U, Vandenabeele P, CLAUSS M. The endothelial monocyte-activating polypeptide II (EMAP II) is a substrate for caspase-7. FEBS LETTERS. 2000;466(1):143–7.
MLA
BEHRENSDORF, HA, Marc Van De Craen, UE KNIES, et al. “The Endothelial Monocyte-activating Polypeptide II (EMAP II) Is a Substrate for Caspase-7.” FEBS LETTERS 466.1 (2000): 143–147. Print.