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Lipopolysaccharide regulates macrophage fluid phase pinocytosis via CD14-dependent and CD14-independent pathways.

MP PEPPELENBOSCH, Marjory Desmedt, T TEN HOVE, SJH VAN DEVENTER and Johan Grooten UGent (1999) BLOOD. 93(11). p.4011-4018
Please use this url to cite or link to this publication:
author
organization
year
type
journalArticle
publication status
published
subject
journal title
BLOOD
Blood
volume
93
issue
11
pages
4011-4018 pages
Web of Science type
Article
Web of Science id
000080558500052
ISSN
0006-4971
language
English
UGent publication?
yes
classification
A1
id
113948
handle
http://hdl.handle.net/1854/LU-113948
date created
2004-01-14 13:35:00
date last changed
2012-06-26 14:29:41
@article{113948,
  author       = {PEPPELENBOSCH, MP and Desmedt, Marjory and TEN HOVE, T and VAN DEVENTER, SJH and Grooten, Johan},
  issn         = {0006-4971},
  journal      = {BLOOD},
  language     = {eng},
  number       = {11},
  pages        = {4011--4018},
  title        = {Lipopolysaccharide regulates macrophage fluid phase pinocytosis via CD14-dependent and CD14-independent pathways.},
  volume       = {93},
  year         = {1999},
}

Chicago
PEPPELENBOSCH, MP, Marjory Desmedt, T TEN HOVE, SJH VAN DEVENTER, and Johan Grooten. 1999. “Lipopolysaccharide Regulates Macrophage Fluid Phase Pinocytosis via CD14-dependent and CD14-independent Pathways.” Blood 93 (11): 4011–4018.
APA
PEPPELENBOSCH, MP, Desmedt, M., TEN HOVE, T., VAN DEVENTER, S., & Grooten, J. (1999). Lipopolysaccharide regulates macrophage fluid phase pinocytosis via CD14-dependent and CD14-independent pathways. BLOOD, 93(11), 4011–4018.
Vancouver
1.
PEPPELENBOSCH M, Desmedt M, TEN HOVE T, VAN DEVENTER S, Grooten J. Lipopolysaccharide regulates macrophage fluid phase pinocytosis via CD14-dependent and CD14-independent pathways. BLOOD. 1999;93(11):4011–8.
MLA
PEPPELENBOSCH, MP, Marjory Desmedt, T TEN HOVE, et al. “Lipopolysaccharide Regulates Macrophage Fluid Phase Pinocytosis via CD14-dependent and CD14-independent Pathways.” BLOOD 93.11 (1999): 4011–4018. Print.