Franky Van Herreweghe
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MK2 phosphorylation of RIPK1 regulates TNF-mediated cell death
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TRAIL induces necroptosis involving RIPK1/RIPK3-dependent PARP-1 activation
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Tumor necrosis factor-mediated cell death: to break or to burst, that's the question
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The role of the kinases RIP1 and RIP3 in TNF-induced necrosis
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Necrosis: molecular mechanisms and physiological roles
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Death receptor-induced necroptosis
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Tumor necrosis factor modulates glycolysis, the glyoxalase system and induces the formation of specific methylglyoxal-modified proteins in aortic endothelial cells
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Methylglyoxal suppresses TNF-alpha-induced NF-kappa B activation by inhibiting NF-kappa B DNA-binding
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A novel cell death pathway as potential therapeutic target for the treatment of cancer
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Tumor necrosis factor-induced modulation of glyoxalase I activities through phosphorylation by PKA results in cell death and is accompanied by the formation of a specific methyl glyoxal-derived AGE.
(2002) PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA. 99(2). p.949-954