prof. dr. Anje Cauwels
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TLR-2 and TLR-9 are sensors of apoptosis in a mouse model of doxorubicin-induced acute inflammation
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Nitrite regulation of shock
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Reactive oxygen species and small-conductance calcium-dependent potassium channels are key mediators of inflammation-induced hypotension and shock
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Nitrite protects against morbidity and mortality associated with TNF- or LPS-induced shock in a soluble guanylate cyclase-dependent manner
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sGCα1β1 attenuates cardiac dysfunction and mortality in murine inflammatory shock models
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- Journal Article
- A1
- open access
Is there NO treatment for severe sepsis?
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- Journal Article
- A1
- open access
IL-17 produced by Paneth cells drives TNF-induced shock
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Critical role for small and large conductance calcium-dependent potassium channels in endotoxemia and TNF toxicity
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Excessive endogenous systemic no production is not sufficient to cause shock, morbidity or mortality
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Nitric oxide in shock