- ORCID iD
- 0000-0002-3212-5516
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- Journal Article
- open access
Proteins à la carte: riboproteogenomic exploration of bacterial N-terminal proteoform expression
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- Journal Article
- A1
- open access
Myb overexpression synergizes with the loss of Pten and is a dependency factor and therapeutic target in T-cell lymphoblastic leukemia
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- Journal Article
- A1
- open access
A positive feedback loop regulation between NOTCH1 and USP11 in T-cell leukemia
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- Journal Article
- A1
- open access
Exposing the small protein load of bacterial life
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- Journal Article
- A1
- open access
Oncogenic deubiquitination controls tyrosine kinase signaling and therapy response in acute lymphoblastic leukemia
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- Journal Article
- A1
- open access
Selective ribosome profiling reveals a role for SecB in the co-translational inner membrane protein biogenesis
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Splicing dysregulation in human hematologic malignancies : beyond splicing mutations
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- Journal Article
- A2
- open access
Hidden in plain sight : challenges in proteomics detection of small ORF-encoded polypeptides
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- Journal Article
- A1
- open access
Small protein enrichment improves proteomics detection of sORF encoded polypeptides
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- Journal Article
- A1
- open access
Capturing salmonella SspH2 host targets in virus-like particles
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- Journal Article
- A1
- open access
Lost and found : re-searching and re-scoring proteomics data aids genome annotation and improves proteome coverage
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- Journal Article
- A1
- open access
Bacterial riboproteogenomics : the era of N-terminal proteoform existence revealed
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The Lrp4R1170Q homozygous knock-in mouse recapitulates the bone phenotype of sclerosteosis in humans
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Genetic control of bone mass
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A novel domain-specific mutation in a sclerosteosis patient suggests a role of LRP4 as an anchor for sclerostin in human bone
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Further delineation of facioaudiosymphalangism syndrome : description of a family with a novel NOG mutation and without hearing loss
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Eight mutations including 5 novel ones in the COL1A1 gene in Czech patients with osteogenesis imperfecta
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Variation in the Kozak sequence of WNT16 results in an increased translation and is associated with osteoporosis related parameters
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Sclerosing bone dysplasias : leads toward novel osteoporosis treatments
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Mutations in sFRP1 or sFRP4 are not a common cause of craniotubular hyperostosis
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The role of extracellular modulators of canonical Wnt signaling in bone metabolism and diseases