- ORCID iD
- 0009-0008-0856-4333
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The impact of hepatocyte-specific deletion of hypoxia-inducible factors on the development of polymicrobial sepsis with focus on GR and PPARα function
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- Journal Article
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Hepatic peroxisome proliferator-activated receptor alpha dysfunction in porcine septic shock
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Point mutation I634A in the glucocorticoid receptor causes embryonic lethality by reduced ligand binding
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Reprogramming of glucocorticoid receptor function by hypoxia
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Combined glucocorticoid resistance and hyperlactatemia contributes to lethal shock in sepsis
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- Journal Article
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ZBTB32 performs crosstalk with the glucocorticoid receptor and is crucial in glucocorticoid responses to starvation
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Zinc inhibits lethal inflammatory shock by preventing microbe-induced interferon signature in intestinal epithelium
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Glucocorticoids limit lipopolysaccharide-induced lethal inflammation by a double control system
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Hepatic PPARα function and lipid metabolic pathways are dysregulated in polymicrobial sepsis
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- Journal Article
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A study of cecal ligation and puncture-induced sepsis in tissue-specific tumor necrosis factor receptor 1-deficient mice