prof. Mathieu Bertrand
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- Journal Article
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Molecular crosstalk between apoptosis, necroptosis, and survival signaling
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- Journal Article
- A1
- open access
Essential versus accessory aspects of cell death: recommendations of the NCCD 2015
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- Journal Article
- A1
- open access
Endoplasmic reticulum stress induces ligand-independent TNFR1-mediated necroptosis in L929 cells
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IAPs, regulators of innate immunity and inflammation
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Type III collagen is important for type I collagen fibrillogenesis and for dermal and cardiovascular development
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NF-κB-independent role of IKKα/IKKβ in preventing RIPK1 kinase-dependent apoptotic and necroptotic cell death during TNF signaling
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NIK promotes tissue destruction independently of the alternative NF-κB pathway through TNFR1/RIP1-induced apoptosis
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cIAP2 supports viability of mice lacking cIAP1 and XIAP
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Type III collagen is important for collagen fibrillogenesis and for dermal and cardiovascular development
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Caspase-3 and RasGAP: a stress-sensing survival/demise switch
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- Journal Article
- A1
- open access
Depletion of RIPK3 or MLKL blocks TNF-driven necroptosis and switches towards a delayed RIPK1 kinase-dependent apoptosis
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- Journal Article
- A1
- open access
MLKL compromises plasma membrane integrity by binding to phosphatidylinositol phosphates
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Deficiency in the mitochondrial apoptotic pathway reveals the toxic potential of autophagy under ER stress conditions
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- Journal Article
- A1
- open access
RIPK1 promotes death receptor-independent caspase-8-mediated apoptosis under unresolved ER stress conditions
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RIPK1 ensures intestinal homeostasis by protecting the epithelium against apoptosis
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Type III collagen is important for type I collagen fibrillogenesis and for dermal and cardiovascular development
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RIPK3 contributes to TNFR1-mediated RIPK1 kinase-dependent apoptosis in conditions of cIAP1/2 depletion or TAK1 kinase inhibition
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C-propeptide mutations in procollagen type I and V are associated with endoplasmic reticulum stress-specific unfolded protein responses
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Loss of Maged1 results in obesity, deficits of social interactions, impaired sexual behavior and severe alteration of mature oxytocin production in the hypothalamus
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The role of the IAP E3 ubiquitin ligases in regulating pattern-recognition receptor signalling
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Intermediate domain of receptor-interacting protein kinase 1 (RIPK1) determines switch between necroptosis and RIPK1 kinase-dependent apoptosis
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RIP1 is required for IAP inhibitor-mediated sensitization of childhood acute leukemia cells to chemotherapy-induced apoptosis
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The unfolded protein response at the crossroads of cellular life and death during endoplasmic reticulum stress
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Smac mimetic bypasses apoptosis resistance in FADD- or caspase-8-deficient cells by priming for tumor necrosis factor α-induced necroptosis
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TNF-induced necroptosis in L929 cells is tightly regulated by multiple TNFR1 complex I and II members
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NOD-like receptors and the innate immune system: coping with danger, damage and death
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cIAP1 and TAK1 protect cells from TNF-induced necrosis by preventing RIP1/RIP3-dependent reactive oxygen species production
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The Ripoptosome: death decision in the Cytosol
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Interaction patches of procaspase-1 caspase recruitment domains (CARDs) are differently involved in procaspase-1 activation and receptor-interacting protein 2 (RIP2)-dependent nuclear factor κB signaling
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- Journal Article
- A1
- open access
cIAP1/2 are direct E3 ligases conjugating diverse types of ubiquitin chains to receptor interacting proteins kinases 1 to 4 (RIP1-4)
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- Journal Article
- A1
- open access
Maged1, a new regulator of skeletal myogenic differentiation and muscle regeneration
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ProNGF induces TNF alpha-dependent death of retinal ganglion cells through a p75(NTR) non-cell-autonomous signaling pathway
(2010) PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA. 107(8). p.3817-3822 -
RIP1's function in NF-kappa B activation: from master actor to onlooker
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Cellular inhibitors of apoptosis cIAP1 and cIAP2 are required for innate immunity signaling by the pattern recognition receptors NOD1 and NOD2
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cIAP1 and cIAP2 facilitate cancer cell survival by functioning as E3 ligases that promote RIP1 ubiquitination
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- Journal Article
- A1
- open access
NRAGE, a p75NTR adaptor protein, is required for developmental apoptosis in vivo
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Comparative expression analysis of the MAGED genes during embryogenesis and brain development
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An overview of the MAGE gene family with the identification of all human members of the family