Mathieu Bertrand
work address
Technologiepark Zwijn. 927 VIB west
9052 Zwijnaarde
9052 Zwijnaarde
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- 2015
- Endoplasmic reticulum stress induces ligand-independent TNFR1-mediated necroptosis in L929 cells
- Essential versus accessory aspects of cell death: recommendations of the NCCD 2015
- 2014
- RIPK1 promotes death receptor-independent caspase-8-mediated apoptosis under unresolved ER stress conditions
- Deficiency in the mitochondrial apoptotic pathway reveals the toxic potential of autophagy under ER stress conditions
- RIPK1 ensures intestinal homeostasis by protecting the epithelium against apoptosis
- MLKL compromises plasma membrane integrity by binding to phosphatidylinositol phosphates
- Depletion of RIPK3 or MLKL blocks TNF-driven necroptosis and switches towards a delayed RIPK1 kinase-dependent apoptosis
- Caspase-3 and RasGAP: a stress-sensing survival/demise switch
- 2013
- RIPK3 contributes to TNFR1-mediated RIPK1 kinase-dependent apoptosis in conditions of cIAP1/2 depletion or TAK1 kinase inhibition
- 2012
- Loss of Maged1 results in obesity, deficits of social interactions, impaired sexual behavior and severe alteration of mature oxytocin production in the hypothalamus